Prevention of "low temperature disease" of chickens in autumn and winter

In the process of growth and development in autumn and winter, chickens are susceptible to rickets and rickets, which are often caused by hypothermia. This is a nutritional disease in which the metabolism of calcium and phosphorus in chickens is destroyed during the cold season, resulting in abnormal bone tissue growth. The onset of adult chicken is often referred to as rickets, and the onset of a chick is often referred to as rickets. Regardless of adult chickens or laying hens, their normal growth is greatly affected after the onset of disease. In particular, the egg production of laying hens has dropped sharply and the heads have become smaller and the quality has deteriorated. Therefore, in the autumn and winter cold season, chicken breeding should pay attention to the prevention and treatment of “low temperature disease”.

Symptoms of diseased chickens are mainly characterized by poor growth, loss of appetite, feathery hair, soft-bending, soft ribs, and hard rib junctions with round, bead-like, hard, flat, flat chest that protrudes to both sides. Severe mouth soft, such as rubber, can be bent, feeding difficulties, chicken feet can not stand the pain, most lying on the bed. Both chicks are curved or X-shaped curved, spine bending, chickens are weak.

Prevention (1) The diet should be formulated according to the nutritional standards of the chicken, and sufficient calcium, phosphorus and vitamin D should be given. It is best to use the full-price compound feed as a condition to ensure that the chicken needs calcium and phosphorus. (2) The farm should provide sufficient sunlight (invalid sunlight passing through ordinary glass), and it is generally advisable to take 16 hours of sunshine per day, and the available electric lighting for lack of sunshine should be used instead. The best condition is to let chickens on the beach. Sunbathing; Closed chicken farms should strengthen the exposure of fluorescent lamps, an increase of 10 minutes per week is appropriate, to maintain a constant. At the same time, gravel is supplemented to increase the digestibility of feed for chickens. (3) Supplementing some green feed or silage with appropriate amount of feed containing vitamins (especially vitamin D), such as cabbage leaves, carrots, and yam leaves.

Treatment (1) When diseased chickens are found, the proportion of calcium and phosphorus in the diet should be adjusted in time. Under normal circumstances, calcium, phosphorus in the diet content: 0.9% of chickens, 1.1% of growing chickens, 3.1% -3.5% of layer chickens, meat chickens, about 0.9%. The ratio of calcium to phosphorus was 21:1 for chicks, 23:1 for growing chickens, and 7:1 for laying chickens. At the same time, Additives can be added to increase the content of various minerals and vitamins in the diet. In particular, we must pay attention to vitamin D or cod liver oil in order to promote the body's absorption of calcium and phosphorus. In general, vitamin D2000 International Units and 1-2 ml per day of cod liver oil per kilogram of feed are used. (2) Bone meal, shell powder, eggshell powder, stone powder, etc. containing more calcium and phosphorus are added to the diet. Under normal circumstances, bone meal only accounts for 1%-2% of the diet, stone powder and shell powder together account for 1%-2% in the chicken's feed, and 2% of the layers and breeder's diet. (3) Severe chickens can be injected subcutaneously with vitamin D injection. Each diseased chicken per day per kilogram body weight 6000-10000 international units, repeated every 3-5 days, generally 2-3 times can be effective, such as with oral calcium better.

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